What is Canine Copper Toxicosis?
Canine Copper Toxicosis is similar to Wilson's Disease in humans: a condition whereby the liver is unable to metabolise copper, which is in many foods and which normally is a crucial part of the diet. Over time, and with build-up of copper, the liver will become scarred, and 'shunts' will form (a by-pass which the blood flow finds for itself.) Scar tissue and inflammation build up and prevent efficient liver function. The liver is an amazing organ that can function on very little of its capacity, and so the condition can be quite advanced before any symptoms are apparent. |
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Medications
Different approaches may be applied depending on the severity of the dog's condition.
Penicillamine is the most commonly given drug; this attracts copper and allows it to be eliminated from the system. A good quality milk thistle supplement is also commonly used: this supports the liver in its self-repair. Steroids may also be prescribed to prevent inflammation. If the dog's condition is advanced, a diuretic, antibiotics, and laxatives may also be used. Vets have differing attitudes to supplements, but there are some that are widely used: Turmeric, with its anti-inflammatory properties, is most effectively used in conjunction with black pepper and coconut oil (forming 'golden paste'). This can be added to food or given on its own. Vitamins B6 and E are widely used. A probiotic such as Lambert's Xtra4 can be used to support the gut. Zinc Acetate is commonly used to manage the condition, but only when treatment with penicillamine is finished, as the two are contraindicated. |
Latest Research
There is one gene which is known to carry CT, and tests are available for this.
It is also suspected that there is a second gene which may be responsible, but no test is yet available. |